during the lc trial, mcr in t subjects was 64% higher than in ut (p < 0.05), in whom %vìo2max and [lactate]b were similar. hence, our first aim of the present study was to interrogate the physiological significance of the lt by determining lactate kinetics in subjects exercising at and just below the lt to test the hypothesis that the lt represents a limitation in lactate clearance. on a subsequent day, subjects underwent an exercise stress test in accordance with the american college of sports medicine guidelines for exercise testing and prescription (7th ed.). blood pressure was measured in the mid stage; rpe was recorded, and a 1-ml blood sample was drawn at the end of each exercise stage. at the onset of exercise, [3-13c]lactate infusion rate was increased to 11.25 mg/min and 15 mg/min for ut and t subjects, respectively. arterialized blood samples (â¼3 ml) for analyses of lactate and glucose concentrations and isotopic enrichments (ie) were collected in 8% perchloric acid in a 1:2 ratio. calculations of lactate kinetics were performed during the last 30 min of rest and 20 min of exercise. figure 1 shows typical blood lactate evolution curves during the incremental exercise used to determine the lt. lt-associated vìo2 and percentage of vìo2max (%vìo2max) were significantly higher in trained cyclists compared with untrained subjects (p < 0.05). to elevate [lactate]b to lt levels in t subjects during the lt-10%+lc trial, mean exogenous lactate infusion rates were 3.91 â± 0.26 mgâ·kgâ1â·minâ1 and 3.16 â± 0.95 mgâ·kgâ1â·minâ1 during rest and exercise, respectively.
blood [glucose] during exercise in lt-10% and lt-10%+lc trials in t subjects was not different from that during lt in ut subjects (21). during the lt-10%+lc trial, ra (28.4 â± 2.8 mgâ·kgâ1â·minâ1) was higher than that during exercise at lt in ut subjects (p < 0.05) and not significantly different from ra at lt in t subjects (ns). during the lt-10%+lc trial, rd (28.4 â± 2.8 mgâ·kgâ1â·minâ1) was higher than that during exercise at lt in ut subjects (p < 0.05) and not significantly different from rd at lt in t subjects (ns). 7a, the slope of the regression between lactate ra and [epinephrine] is greater in athletes than in untrained men. both vasoactive and metabolic effects of circulating catecholamines have the potential to affect lactate kinetics. 8 illustrates the relationship between lactate mcr and absolute and relative metabolic rates in the present and former studies involving healthy men of variable fitness levels. the comparison of data obtained during the lt trial in ut subjects and the lt-10% trial in t subjects (both at â¼67% vìo2max) showed the effects of long-term endurance training on lactate kinetics during exercise of similar relative intensity. as a result, mcr remained significantly higher by 64% (p < 0.05) in t subjects than in ut subjects for the same relative (i.e., higher absolute) exercise intensity and similar [lactate]b. in the aggregate, the present results (figs. we used combinations of exercise intensity and a lactate clamp technique involving exogenous lactate infusion to interrogate the meaning of the lt in healthy young men and competitive male cyclists. in contrast to the comparison in trained men exercising at lt and a power output lesser by 10%, exogenous lactate infusion resulted in an increase in lactate disposal.
to understand the meaning of the lactate threshold (lt) and to test the hypothesis that endurance ra was not different between ut and t when relative exercise intensities were matched (ut-lt vs. vo2 max, lactate threshold (lt) and vo2 at lt (vo2lt) among aerobic trained athletes and untrained participants. simon et al (76) demonstrated that in sedentary males, ventilatory threshold (51% of vo, what is the lactate threshold, what is the lactate threshold, lactate threshold training, anaerobic threshold vs lactate threshold, how lactate threshold training can impact aerobic performance.. steady-state training at the lactate threshold is often referred to as \u201cmaximal steady-state\u201d exercise or \u201ctempo runs.\u201d research has shown that the lactate threshold occurs at 80-90% of heart rate reserve (hrr) in trained individuals and at 50-60% hrr in untrained individuals (weltman 1995).
max) occurred significantly before blood lactate threshold (62% vo max), whereas, in trained males the onset of ventilatory threshold (66% vo max) was not different from blood lactate threshold (69% of vo, what factors influence the percent of vo2max at which the lactate threshold occurs?, how does lactate threshold related to aerobic and anaerobic metabolism, lactate threshold 1 and 2, lactate threshold graph
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